Pro-Vaccine Video by Monica Gomez

These pro-vaccine arguments are from Carrington College in America. http://carrington.edu/blog/medical/vaccines/vaccines-work/

This page was requested by Monica Gomez.

How Do Vaccines Work?

What is a vaccine? A vaccine is a biological preparation that improves immunity to a particular disease. It contains an agent resembling a disease-inducing microorganism– a bacterium, virus or toxin – that activates the body’s immune system. White blood cells – APCs, B cells, and T-cells – recognize, destroy and “remember” this version of the pathogen. That way, the immune system can quickly recognize and destroy this harmful microorganism later on. A vaccine is essentially a pathogen-imposter.
Today, there are five main types of vaccines. Live, attenuated vaccines fight viruses and contain a weakened version of the living virus (e.g., measles-mumps-rubella and varicella vaccine). Inactivated vaccines also fight viruses and contain the killed virus (e.g., polio vaccines). Toxoid vaccines prevent diseases caused by bacteria that produce toxins in the body and contain weakened toxins (e.g., diphtheria and tetanus vaccine). Subunit vaccines include only the essential antigens of the virus or bacteria (e.g. whooping cough vaccine). Conjugate vaccines fight a different type of bacteria which have antigens with an outer coating of sugar-like substances (polysaccharides) that “hide” the antigen from the child’s immature immune system; the vaccine connects (conjugates) the polysaccharides to antigens, so the immune system can react.

A vaccine is essentially a pathogen-imposter.

Once the altered pathogen is introduced into the bloodstream, it is captured by antigen-presenting cell (APC), which float around looking for invaders. When an APC detects the vaccine antigen, it ingests it, breaks it apart, and displays a piece of the antigen on its surface. Then, it travels to areas where immune cells cluster (e.g. lymph nodes) and where so-called naïve T cells specific to the antigen recognize it as foreign and become activated. These T helper cells alert other nearby cells. Naïve B cells recognize the antigens carried by the APCs as well and also become activated.
Some naïve B cells mature into plasma B cells after activation by vaccine antigens and reception of signals from activated helper T cells. They produce antibodies which are “y” shaped proteins that are released at high levels every second. Each antibody tightly attaches to a specific target antigen (like a lock and a key), which can prevent the antigen from entering a cell or mark the antigen for destruction. If the vaccine contains weakened viruses, they enter the cells which are then killed by Killer T cells. What follows is the development of memory (B, T helper and killer T) cells that memorize the vaccine antigen and recognize the real pathogen in the future.
This means that the body’s response will be stronger and faster than if it had never encountered the pathogen before. This is called a secondary response to the pathogen. Furthermore, secondary responses will result in the production of more antibodies to fight the pathogen and more memory cells to identify it promptly. Thus, vaccinations “program” the immune system to remember a specific disease-inducing microorganism by letting it “practice” with a weakened, killed or inactivated version of the pathogen.

Vaccines can prevent outbreaks of contagious diseases through herd immunity (or community immunity). This means that a sufficient portion of the population must be immune to an infectious disease (by vaccination and/or prior illness), so that the disease is less likely to spread from one person to another. As the number of vaccinated people increases, the protective effect of herd immunity increases as well. While the herd immunity threshold may start with 40% of the population vaccinated for some diseases, most diseases require vaccination rates as high as 80% – 95% to prevent outbreaks. Moreover, herd immunity protects those who cannot be vaccinated or for whom the vaccination was not successful, such as people with weak immune systems, chronic illnesses or allergies.

VAN UK’s Response:

Vaccinations induce antibodies by a T cell Helper 2 response (TH2). Antibodies are thought to protect the individual from parasites, toxins and other ‘outside’ environmental exposures. This is known as the humoral response.
TH2 is an anti-inflammatory, suppressive component of the immune system and is the dominant part of the immune system that is at work during a mother’s pregnancy. Although it makes antibodies, it doesn’t react with an inflammatory response that could potentially damage the developing fetus. The neonates passive immunity is therefore TH2 based.
There is also a T helper 1 (TH1) response, known as the cellular response which helps to bring about immunity within our cells to viruses, yeast, cancer by inducing an inflammatory response from the cells. TH1 response stimulates skin reactions to diseases (inflammation, rashes etc) and hypersensitivity reactions. We have both TH1 and TH2 to balance out our immune response so that we can produce antibodies to infection without incuring too much inflammation and tissue damage.

The British Medical Journal wrote:

‘Th1-type cytokines tend to produce the proinflammatory responses responsible for killing intracellular parasites and for perpetuating autoimmune responses. Interferon gamma is the main Th1 cytokine. Excessive proinflammatory responses can lead to uncontrolled tissue damage, so there needs to be a mechanism to counteract this. The Th2-type cytokines include interleukins 4, 5, and 13, which are associated with the promotion of IgE and eosinophilic responses in atopy, and also interleukin-10, which has more of an anti-inflammatory response. In excess, Th2 responses will counteract the Th1 mediated microbicidal action. The optimal scenario would therefore seem to be that humans should produce a well balanced Th1 and Th2 response, suited to the immune challenge.’

Put basically, we need a balanced amount of T cell 1 and 2 in order to have a healthily functioning immune system.

What vaccination does is over-stimulate the TH2 immune system, which simultaneously suppresses TH1. This causes vaccinated individuals to become hypersensitive to toxins, allergens and bacteria while not responding to viruses, yeast and cancer. This is a large reason why we now have 1 in 3 people who get cancer and an epidemic of children with asthma, eczema, hayfever and food allergies.

BMJ also wrote:

‘Many researchers regard allergy as a Th2 weighted imbalance, and recently immunologists have been investigating ways to redirect allergic Th2 responses in favour of Th1 responses to try to reduce the incidence of atopy.’

Their investigations involve developing more vaccines rather than just letting the immune system function as mother nature designed it to.

This over-activation of TH2 gives us a hypersensitive immune system that over-reacts to bacteria, toxins and environmental pollutants, increasing the likelihood of eczema, asthma, hayfever, RH arthritis, MS, type 1 diabetes, food allergies and other inflammatory diseases. The down-regulation of TH1 means that the immune system will under-respond to challenges in the cells, for instance, cancer and yeast. In addition to crazy cancer levels, many people have chronic yeast problems such as vulvodynia, intersital cystitis, recurrent candida infections, UTI’s, gum infections and more.

Depression and anxiety have also been on the increase in recent decades and it isn’t just because of greater awareness. Recent discoveries have found that inflammation can trigger anxious and depressed behaviours.
In 2008, researchers used BCG vaccine to induce anxious behaviour in rats. Brain, behaviour and immunity wrote:

‘Although cytokine-induced sickness behavior is now well-established, the mechanisms by which chronic inflammation and depression are linked still remain elusive. Therefore this study aimed to develop a suitable model to identify the neurobiological basis of depressive-like behavior induced by chronic inflammation, independently of sickness behavior. We chose to measure the behavioral consequences of chronic inoculation of mice with Bacillus Calmette-Guerin (BCG), which has been shown to chronically activate both lung and brain indoleamine 2,3-dioxygenase (IDO), a tryptophan-catabolizing enzyme that mediates the occurrence of depressive-like behavior following acute innate immune system activation. BCG inoculation induced an acute episode of sickness (approximately 5 days) that was followed by development of delayed depressive-like behaviors lasting over several weeks. Transient body weight loss, reduction of motor activity and the febrile response to BCG were dissociated temporarily from a sustained increase in the duration of immobility in both forced swim and tail suspension tests, reduced voluntary wheel running and decreased preference for sucrose (a test of anhedonia). Moreover, we show that a distinct pattern of cytokine production and IDO activation parallels the transition from sickness to depression. Protracted depressive-like behavior, but not sickness behavior, was associated with sustained increase in plasma interferon-gamma and TNF-alpha concentrations and peripheral IDO activation. Together, these promising new data establish BCG inoculation of mice as a reliable rodent model of chronic inflammation-induced depressive-like behaviors that recapitulate many clinical observations and provide important clues about the neurobiological basis through which cytokines may have an impact on affective behaviors.’

So if you have a family history of auto-immune disease, allergies or cancer and you have depression or anxiety that isn’t due to a stressful situation, you are probably chronically inflammed.
Vaccination doesn’t strengthen the immune system, it skews it towards a TH2 response and this imbalance causes disease.

When vaccines were invented, no one knew anything about the TH1 and TH2 responses. Vaccines today are based on humoral (antibody) immunity – the theory that antibodies are produced when the body encounters a pathogen it has experienced before. However, this theory has quite a few holes.
In the 1940’s, immunologist Merill Chase, discovered that it was not antibodies alone that brought immunity.

New York Times wrote:

‘Dr. Merrill W. Chase, was an immunologist whose research on white blood cells helped undermine the longstanding belief that antibodies alone protected the body from disease and micro-organisms. Dr. Chase made his landmark discovery in the early 1940’s while working with Dr. Karl Landsteiner, a Nobel laureate recognized for his work identifying the human blood groups. At the time, experts believed that the body mounted its attacks against pathogens primarily through antibodies circulating in the blood stream, known as humoral immunity.
But Dr. Chase, working in his laboratory, stumbled upon something that appeared to shatter that widespread tenet.
As he tried to immunize a guinea pig against a disease using antibodies he had extracted from a second pig, he found that blood serum did not work as the transfer agent.
Not until he used white blood cells did the immunity carry over to the other guinea pig, providing solid evidence that it could not be antibodies alone orchestrating the body’s immune response.’

Nature Immunology make a surprisingly frank admission, in over 200 years of vaccine ‘science’ scientists still don’t understand the exact mechanisms of vaccines or the immune system:
‘Despite their success, one of the great ironies of vaccinology is that the vast majority of vaccines have been developed empirically, with little or no understanding of the immunological mechanisms by which they induce protective immunity…… the failure to develop vaccines against global pandemics such as infection with human immunodeficiency virus (HIV) despite decades of effort has underscored the need to understand the immunological mechanisms by which vaccines confer protective immunity. It is now clear that the immune system has evolved qualitatively different types of responses to protect against different pathogens.’

Today, vaccines are still based upon antibody response, with antibody count levels being associated with immunity from disease, but there have been lots of cases in which people with high antibody titers have still got the disease and this may be why there are still frequent epidemics in vaccinated people.
For instance, this case report in Neurology details the cases of three individuals who were vaccinated against tetanus and had high antibody counts but still developed tetanus anyway:

‘Severe (grade III) tetanus occurred in three immunized patients who had high serum levels of anti-tetanus antibody. The disease was fatal in one patient. One patient had been hyperimmunized to produce commercial tetanus immune globulin. Two patients had received immunizations 1 year before presentation. Anti-tetanus antibody titers on admission were 25 IU/ml to 0.15 IU/ml by hemagglutination and ELISA assays; greater than 0.01 IU/ml is considered protective. Even though one patient had seemingly adequate anti-tetanus titers by in vitro measurement (0.20 IU), in vivo mouse protection bioassays showed a titer less than 0.01 IU/ml, implying that there may have been a hole in her immune repertoire to tetanus neurotoxin but not to toxoid. This is the first report of grade III tetanus with protective levels of antibody in the United States. The diagnosis of tetanus, nevertheless, should not be discarded solely on the basis of seemingly protective anti-tetanus titers.’

In an outbreak of mumps at a university where more than 95% of the students had had two doses of MMR, antibody titer levels between those affected and those unaffected were similar between the two groups:

‘BACKGROUND:

In 2006, a mumps outbreak occurred on a university campus despite ≥ 95% coverage of students with 2 doses of measles-mumps-rubella (MMR) vaccine. Using plasma samples from a blood drive held on campus before identification of mumps cases, we compared vaccine-induced preoutbreak mumps antibody levels between individuals who developed mumps (case patients) and those who did not develop mumps (nonpatients).
METHODS:

Preoutbreak samples were available from 11 case patients, 22 nonpatients who reported mumps exposure but no mumps symptoms, and 103 nonpatients who reported no known exposure and no symptoms. Antibody titers were measured by plaque reduction neutralization assay using Jeryl Lynn vaccine virus and the outbreak virus Iowa-G/USA-06 and by enzyme immunoassay (EIA).

RESULTS:

Preoutbreak Jeryl Lynn virus neutralization titers were significantly lower among case patients than unexposed nonpatients (P = .023), and EIA results were significantly lower among case patients than exposed nonpatients (P = .007) and unexposed nonpatients (P = .009). Proportionately more case patients than exposed nonpatients had a preoutbreak anti-Jeryl Lynn titer < 31 (64% vs 27%, respectively; P = .065), an anti-Iowa-G/USA-06 titer < 8 (55% vs 14%; P = .033), and EIA index standard ratio < 1.40 (64% vs 9%; P = .002) and < 1.71 (73% vs 14%, P = .001).

DISCUSSION:

Case patients generally had lower preoutbreak mumps antibody levels than nonpatients. However, titers overlapped and no cutoff points separated all mumps case patients from all nonpatients.’

‘Immune’ in vaccination terms means that after a certain period of time, antibody levels are still high, but they don’t know what level constitutes immunity and they don’t follow up the vaccinated in the long-term to find out if they got any of the diseases they were vaccinated for.

There’s also contradiction over what antibodies actually mean. If a child has antibodies to whooping cough or polio they are said to be immune, but if they have antibodies to tuberculosis or HIV they are said to be infected. The HIV test, for example, looks for antibodies in the blood and if you’ve got them you are diagnosed as HIV Positive. It could be that the presence of antibodies just means that you have been exposed to an illness, rather than you are actually immune to it.

Scientists have recently discovered that you don’t even need antibodies to become immune to some serious infections when they infected mice with vesicular stomatitis virus (VSV). The mice produced B cells but not antibodies. While the B cells were essential, the mice didn’t need antibodies in order to survive the exposure:

‘Neutralizing antibodies have been thought to be required for protection against acutely cytopathic viruses, such as the neurotropic vesicular stomatitis virus (VSV). Utilizing mice that possess B cells but lack antibodies, we show here that survival upon subcutaneous (s.c.) VSV challenge was independent of neutralizing antibody production or cell-mediated adaptive immunity. However, B cells were absolutely required to provide lymphotoxin (LT) α1β2, which maintained a protective subcapsular sinus (SCS) macrophage phenotype within virus draining lymph nodes (LNs). Macrophages within the SCS of B cell-deficient LNs, or of mice that lack LTα1β2 selectively in B cells, displayed an aberrant phenotype, failed to replicate VSV, and therefore did not produce type I interferons, which were required to prevent fatal VSV invasion of intranodal nerves. Thus, although B cells are essential for survival during VSV infection, their contribution involves the provision of innate differentiation and maintenance signals to macrophages, rather than adaptive immune mechanisms.’

Sources:
Immunity. 2012 Mar 23;36(3):415-26. doi: 10.1016/j.immuni.2012.01.013. Epub 2012 Mar 1.
J Infect Dis. 2011 Nov;204(9):1413-22. doi: 10.1093/infdis/jir526. Epub 2011 Sep 20
Nat Immunol. 2011 Jun; 12(6): 509–517.
Brain Behav Immun. 2008 Oct;22(7):1087-95. doi: 10.1016/j.bbi.2008.04.001. Epub 2008 May 13
BMJ 2000;321:424
http://www.altmedrev.com/publications/8/3/223.pdf
Front Neurosci. 2015 Feb 12;9:16. doi: 10.3389/fnins.2015.00016. eCollection 2015.

Neurology. 1992 Apr;42(4):761-4

TH1/TH2 Balance

Th1 and Th2 responses: what are they?

Inoculation of Bacillus Calmette-Guerin to mice induces an acute episode of sickness behavior followed by chronic depressive-like behavior.

Immunological Mechanisms of Vaccination

Severe tetanus in immunized patients with high anti-tetanus titers.

Mumps antibody levels among students before a mumps outbreak: in search of a correlate of immunity.

B cell maintenance of subcapsular sinus macrophages protects against a fatal viral infection independent of adaptive immunity.

Antibodies Alone Do Not

Evidence suggests that the Chinese used smallpox inoculation as early as 1000 BC

Vaccinations are essentially prophylactic, although there has been an effort in recent years to develop therapeutic vaccinations for infectious diseases like AIDS, tuberculosis, cancer, and various autoimmune diseases. There are also potential vaccinations in development for myasthenia gravis, lupus and diabetes, as well as for cognitive diseases such as Alzheimer’s disease, prion diseases and Huntington’s disease.

Usually, vaccinations are administered in the form of an injection into the skin or a liquid taken orally. However, some vaccinations may also be processed by inhalation through mouth/nose or application onto the skin. The vaccines risks are very low. Most vaccine reactions are usually minor and temporary (i.e. sore arm, fatigue or an elevated temperature). Very serious side effects like severe allergic reactions are extremely rare and are carefully monitored and investigated. The vaccine benefits definitely outweigh the vaccine dangers. In fact, it is far more likely to be seriously harmed by a vaccine-preventable disease than by the vaccine itself.

VAN UK’s Response:

It is true that there were superstitious practices among ancients who used to introduce disease to a wound in order to try and ward off illness, and this may be where the founders of vaccination got their idea from. However, the first organised vaccination wasn’t carried out until 1721 by a reverand called Coton Mather. Rev. Mather believed in witches and was responsible for trying women in court and condemning them to burn. This was his rationality and science.

Also, the vaccine used was never smallpox, but cowpox from cattle – an entirely different disease. Despite them being too different disease entities, the claim is still made to this day that the ‘smallpox’ vaccine containing cowpox prevented smallpox.

The initial efforts at vaccination – called variolation – involved cutting the patient and putting festering cowpox into their bleeding arm. As you can imagine, many people died (the vaccinator’s estimations were 2% but it was likely to be much higher) and there was spread of disease. Because of this, there was widespread opposition to it. In fact, Rev. Mather’s entire congregation revolted against him and rioted and one of his disgruntled parishoners put a BOMB through his letterbox, with a note saying ‘Dam you, I’ll innoculate you with this!”

It was the same in England with increases in disease and deaths wherever it was performed and variolation was BANNED under threat of imprisonment by the British Parliament in 1840.

Leicester largely rejected vaccination when the towns people saw what it did to the recipients and they adopted a policy of isolation instead. They had one of the lowest death rates from smallpox in the UK.

So clearly the picture wasn’t as pretty as they like to paint. They can throw around how many lives they estimate that vaccines save but if you look at statistics from earlier times, they don’t back up the theory that vaccines reduce mortality. Mr Wakley, Editor of The Lancet medical journal in 1840, asserted that the great number of deaths from smallpox were actually due to vaccination.

Doctor Hadwen said in a speech in Goddard’s Rooms, Gloucester UK, on January 25th, 1896:

‘I will give you one or two statistics with regard to Leicester. In 1868-72 the mortality of children under one year was 107 per thousand, when 98 per cent were vaccinated; from 1888-9 only two per cent, were vaccinated, and, in spite of what Dr. Bond says, the general mortality of children had declined from 107 to 63 per thousand. Furthermore, from 1874-89 the number of children under one year who died of erysipelas had declined from 193 to 47 per 10,000 deaths. The Guardians of Gloucester are being urged to re-commence prosecutions, and I appeal to them to make a firm stand against it.” – (Cheers erupted in the hall from the audience when he said this).

The act of vaccination in those days could also spread blood bourne diseases as it was eve less sterile than today so if children didn’t get smallpox they would frequently die of sepsis or even syphilis – the bone ravaging sexually transmitted disease that was previously spread by prostitution. Dr Hadwin said:

‘It is a very strange thing that up to 1853, when the Compulsory Vaccination Act was passed, the annual deaths from syphilis of children under one year old did not, exceed 380; the very next year the number had jumped up nearly double, to 591 ; and syphilis in infants under one year of age has gone on increasing every year since until 1883, when the number of deaths reached 1,813. It has increased four-fold in infants since the passing of the Compulsory Vaccination Act, and yet in adults it has remained almost stationary.”

The fact that cases among adults had not increased was proof that vaccination was the cause as there was no other way that an infant could obtain it.

There is very little evidence that vaccination had anything to do with a decline in mortality from infectious disease and despite claims there has been limited study on their effect. The BMJ wrote in:

‘Vaccine programmes must consider their effect on general resistance’
‘Surprisingly, therefore, there are few observational studies and virtually no randomised clinical trials documenting the effect on child mortality of any of the existing vaccines.’

The same is true for other countries. McKinlay said in ‘The Questionable Contribution of Medical Measures to the Decline of Mortality in the United States in the twentieth Century’:

‘In general, medical measures (both chemotherapeutic and prophylactic) appear to have contributed little to the overall decline in mortality in the United States since about 1900, having in many instances been introduced several decades after a marked decline had already set in and having no detectable influence in most instances.’

As far as developing new vaccines for diseases like cancer, lupus and diabetes – these are all diseases that can be triggered by vaccination through the skewing of the TH1/TH2 response and by the introduction of foreign substances into the body. The fact that these diseases are rampant in our modern society may be in part due to the ever increasing number of vaccinations we are expected to have.

The North American Journal of Medical Sciences wrote:

‘Under normal circumstances exposure to a viral disease would be countered (in vivo) at various levels enabling the body to steadily increase its immune response. By contrast, the injection of vaccines directly into the blood system overpowers the normal immune response leading to its rapid depletion. It is now suspected that long-term persistence of viruses and other proteins may produce chronic disease i.e. instead of producing a genuine immunity the vaccines are altering the body’s systemic and biochemical stability, suppressing the production of differing types of white blood cells and hence immune function. Furthermore the introduction of many vaccines (up to 30 in a typical vaccination schedule) introduces a large number of foreign proteins which may be sufficient to ensure that immune function never returns to baseline and/or that immune biochemistry is fundamentally altered[62]. Consequently there now exists a growing concern which links immunizations to the huge increase in recent decades of auto-immune diseases[79] e.g., rheumatoid arthritis[80,81], multiple sclerosis, lupus erythematosus, lymphoma, leukemia, autoimmune demyelinative optic neuritis, diabetes mellitus, etc.’

Prion diseases like nvCJD have in the past been transmitted to people via vaccination. More than one individual who received the same lot of polio vaccine went on to develop the debilatating brain disease and in 1997 the Italian health ministry banned the HIB vaccine because it was made using bovine tissue that could have been infected. The BMJ wrote:

‘The Italian ministry of health has suspended the use and marketing of a vaccine against Haemophilus influenzae type b (Hib) because of fears that it could transmit bovine spongiform encephalopathy (BSE) to humans.The police were called in to seize batches of HibTITER from the Italian outlets of the US manufacturer Wyeth-Lederle on 17 January. The vaccine was used in the Italian national vaccination programme, but the use of bovine heart-brain infusion agar to promote bacterial growth early in the manufacturing process.’

It is much easier to get the disease via blood donation, vaccines and surgery than it is to eat an infected product.

There are a lot more side-effects to vaccination than just a sore arm or a fever! The CDC list these side-effects on their vaccine information statement for DTaP:

DTaP Vaccine
- Mild Problems: Fussiness (up to 1 child in 3); tiredness or loss of appetite (up to 1 child in 10); vomiting (up to 1 child in 50); swelling of the entire arm or leg for 1-7 days (up to 1 child in 30) – usually after the 4th or 5th dose.
- Moderate Problems: Seizure (1 child in 14,000); non-stop crying for 3 hours or longer (up to 1 child in 1,000); fever over 105°F (1 child in 16,000).
- Serious problems: Long term seizures, coma, lowered consciousness, and permanent brain damage have been reported following DTaP vaccination. These reports are extremely rare.
They also say:

‘As with any medicine, there is a very remote chance of a vaccine causing a serious injury or death.’
EMC Medicines lists these side-effects for MMR:

Nasopharyngitis, Upper respiratory tract infection or Viral infection, Aseptic meningitis^†, Atypical measles, Epididymitis, Orchitis, Otitis media, Parotitis, Rhinitis, Subacute Sclerosing Panencephalitis^†,Regional lymphadenopathy, Thrombocytopenia, Anaphylactoid reaction, Anaphylaxis and related phenomenon such as Angioneurotic oedema, Facial oedema, and Peripheral oedema, Irritability, Afebrile convulsions or seizures, Ataxia, Dizziness, Encephalitis^† , Encephalopathy^† , Febrile convulsion (in children), Guillain-Barre syndrome, Headache, Measles inclusion body encephalitis (MIBE) (see section 4.3), Ocular palsies, Optic neuritis, Paraesthesia, Polyneuritis, Polyneuropathy, Retrobulbar neuritis, Syncope, Conjunctivitis, Retinitis, nerve deafness, Rhinorrhoea, Bronchial spasm, Cough, Pneumonia, Pneumonitis, sore throat, Diarrhoea or Vomiting, nausea, Rash morbilliform or other Rash,Urticaria, Panniculitis, Purpura, Skin induration, Stevens-Johnson syndrome, Pruritus, Arthritis^† and/or Arthralgia^† (usually transient and rarely chronic), Myalgia, Fever (38.5°C or higher), Injection site erythema, Injection site pain, and Injection site swelling, Vasculitis.

The data sheet also says:

‘Cases of aseptic meningitis have been reported following measles, mumps, and rubella vaccination. Although a causal relationship between other strains of mumps vaccine and aseptic meningitis has been shown, there is no evidence to link Jeryl Lynn™ mumps vaccine to aseptic meningitis.

In severely immunocompromised individuals inadvertently vaccinated with measles-containing vaccine, measles inclusion body encephalitis, pneumonitis, and fatal outcome as a direct consequence of disseminated measles vaccine virus infection have been reported (see section 4.3); disseminated mumps and rubella vaccine virus infection has also been reported.

There have been reports of SSPE in children who did not have a history of infection with wild-type measles but did receive measles vaccine. Some of these cases may have resulted from unrecognized measles in the first year of life or possibly from the measles vaccination.

Arthralgia and/or arthritis (usually transient and rarely chronic), and polyneuritis are features of infection with wild-type rubella and vary in frequency and severity with age and sex, being greatest in adult females and least in prepubertal children. Following vaccination in children, reactions in joints are generally uncommon (0-3%) and of brief duration. In women, incidence rates for arthritis and arthralgia are generally higher than those seen in children (12-20%), and the reactions tend to be more marked and of longer duration. Symptoms may persist for a matter of months or on rare occasions for years.

The Tripedia DTaP vaccine that has now been discontinued lists autism as a side-effect of the vaccine on their data sheet:

Adverse events reported during post-approval use of Tripedia vaccine include idiopathic thrombocytopenic purpura, SIDS,anaphylactic reaction, cellulitis, autism, convulsion/grand mal convulsion, encephalopathy, hypotonia, neuropathy, somnolence and apnea.’

It is just not accurate or true to suggest that side-effects are only mild when researchers are linking chronic conditions with vaccines, parents are seeing the decline in their children’s health and there are one in six American children with developmental disabilities and 1 in 68 with autism. The manufacturers also freely provide information to the contrary that can be accessed by any parent.

In addition, the reporting system is wholly ineffective. It is voluntary and most doctors don’t report. According to David Kessler, MD, from the FDA, only around 1% of all drug reactions ever get reported. The Vaccine Adverse Events Reporting System (VAERS) is also criticised for taking anecdotal reports and whenever serious events do get reported, those in positions of authority who are supposed to protect public health, merely say they don’t equal causation or are a ‘coincidence.’ Vaccine manufacturers are also sheilded from prosecution because vaccines were ruled ‘unavoidably unsafe’ so there is no motivation for them, the FDA or VAERS to ensure that they are safe.

The multiple vaccination schedule has also never been tested to see if it is actually safe to give a baby up to 7 injections at once and there have never been any large scale trials of vaccinated vs. unvaccinated children to see who is the healthiest. One such trial of TB vaccine was halted in 1979 because children in the vaccinated group died.

There have only been a few studies looking at vaccination’s effect on childhood mortality and most of those showed an increase in mortality with one showing a benefit for pneumonia vaccine so really there is no solid science to back up the claim that vaccines are safe, prevent disease and lower mortality.

So what exactly is the risk to a ‘first world’ child of dying of infectious disease? Let’s look at some of the diseases:

Measles – According to the vaccine manufacturer, Sanofi Pasteur,

‘More than 95% occur in low-income countries with weak health infrastructure.’

That is around 4 ‘first world’ people that die of measles out of every 100 fatal cases. The CDC in fact report that the death rate in the U.S is 0.2% (assuming that statistic isn’t inflated in light of the recent fraud revelations). The CDC say that in 2015 there were 189 measles cases. This means that 188.8 of the people affected did NOT die according to their statistics.

More than half of the total global deaths from measles (164,000 per annum) occur in India so you slash your child’s chances of dying from measles just by not going or living in India.

Surely that’s an argument to vaccinate third world kids? I hear you say. No because according to doctors from India, children who are malnourished and immuno-compromised die from vaccination – it’s why those same children that die from vaccination would probably die from measles if they caught it. Nutrition, sanitation, clean water and vitamin A supplementation is the only real way to stop complications from measles.

Think about it – if you stopped eating fresh fruit and vegetables, if you didn’t have clean running water to wash in or to drink, if you couldn’t be guaranteed a proper sewerage system and you didn’t have reliable electricity to keep you warm, how long do you really think your vaccines would protect you? That’s why there are reports of measles in vaccinated children who live in slums.

Rotavirus – This is a gastroenteritis bug that nearly everychild gets. The vaccine was developed for third world countries where there is often no clean water or access to hospital and without rehydration sachets, babies from poor families can die. In first world countries, rotavirus is caused by lack of breastfeeding, poor sterilization of feeding equipment and not washing hands. In immuno-competant children, it doesn’t kill and is easily prevented by choosing to breastfeed but instead of restricting formula to prescription only to protect babies, doctors don’t tell mothers the truth and would rather sell them a vaccine for something their breast milk would have prevented and that can cause serious side-effects including a fatal bowel disease. 3 or 4 immuno-suppressed children die in the UK every year due to rotavirus.

79 countries in the world have less than 5 rotavirus deaths per year, whereas there were 47,100 deaths per year in India. Only 4 countries (India, Nigeria, Pakistan and the Democratic Republic of the Congo) make up 49% of worldwide deaths in 2013.

Intussusception – a condition where the bowel folds in on itself – is a side-effect of the vaccine. It is fatal unless surgery is quickly performed. In the UK, there are 21 cases of intussusception a year that are directly attributable to the vaccine.

Pertussis – This is a cough that occurs in bouts. It is infectious for 3 weeks but can cause symptoms up to 3 months. In very small babies it can cause problems breathing. Most of the severe complications and deaths are in babies under six months of age. During 2012, a peak year for pertussis, there were 48,277 cases and 20 deaths. According to the VAERS database, in the same year there were 283 deaths from all pertussis containing vaccines:

‘Found 283 cases where Vaccine targets Pertussis (6VAX-F or DPIPV or DPP or DTAP or DTAPH or DTAPHEPBIP or DTAPIPV or DTAPIPVHIB or DTP or DTPHEP or DTPHIB or DTPIHI or DTPIPV or DTPPHIB or PER or TDAP or TDAPIPV) and Patient Died and Vaccination Date on/after ‘2012-01-01’

Which one sounds more dangerous to you?

Polio – There hasn’t been a single case of indiginous wild polio in the U.S since 1979. ALL cases since then were either imported or caused by the vaccine. JAMA wrote:

‘The last case of poliomyelitis in the United States due to indigenously acquired wild poliovirus occurred in 1979; however, as a consequence of oral poliovirus vaccine (OPV) use that began in 1961, an average of 9 cases of vaccine-associated paralytic poliomyelitis (VAPP) were confirmed each year from 1961 through 1989.’

Contrary to popular belief, even when we did have traditional polio (now renamed AFP), the vast majority of cases don’t have any symptoms at all so the person doesn’t even know they are ill – which means you could have already had polio and just got over it. The CDC Pink book says:

‘Up to 72% of all polio infections in children are asymptomatic….

Approximately 24% of polio infections in children consist of a minor, nonspecific illness without clinical or laboratoryevidence of central nervous system invasion. This clinical presentation is known as abortive poliomyelitis, and is characterized by complete recovery in less than a week. This is characterized by a low grade fever and sore throat….Nonparalytic aseptic meningitis (symptoms of stiffness of the neck, back, and/or legs), usually following several days after a prodrome similar to that of minor illness, occurs in 1%–5% of polio infections in children. Increased or abnormal sensations can also occur. Typically these symptoms will last from 2 to 10 days, followed by complete recovery. Fewer than 1% of all polio infections in children result in flaccid paralysis.’

So less than 1% get the paralysis they like to scare parents with, most have NO symptoms, some have something that feels like the flu and some have something resembling viral meningitis (the non-fatal variety).

Despite the fact that we have NO cases of traditional wild polio, in the year 2012, there were 279 deaths reported to VAERS after polio containing vaccines.

‘Found 279 cases where Vaccine targets Polio (6VAX-F or DPIPV or DPP or DTAPHEPBIP or DTAPIPV or DTAPIPVHIB or DTIPV or DTPIHI or DTPIPV or DTPPHIB or IPV or OPV or TDAPIPV) and Patient Died and Vaccination Date on/after ‘2012-01-01’

The Grave Marker of Paleg Conklin, aged 17, who Died of Smallpox By Inoculation on 27th January 1788.