‘We are seeing increasing numbers of small children with hyperacusis. This may be on its own, or in association with other processing or behavioural disorders such as A.D.D., autism and Williams syndrome.’

Here is another example of hyperacusis after live virus vaccine (MMR) in a one year old male child on a VAERS report:

Symptoms: AutismEncephalopathyHyperacusisImmunoglobulins decreasedImmunoglobulins increasedMental retardation severity unspecifiedPersonality disorderSpeech disorder
SMQs:, Dementia (broad), Psychosis and psychotic disorders (broad), Guillain-Barre syndrome (broad), Noninfectious encephalitis (broad), Noninfectious encephalopathy/delirium (narrow), Hostility/aggression (broad), Hearing impairment (narrow) Write-up: Pt recvd MMR vax & exp fever, austistic behaviors, encephalatic condition, began to tune out; sound sensitivity, hand-flapping, wheel-spinning; nightime sweats, appetite inc;

(VAERS ID:42498).

The American Academy of Otolaryngology – Head and Neck Surgery say in their factsheet on hyperacusis that depression may be a factor in people with hyperacusis.  They write this as if it is a cause.  I don’t believe so.  I think that depression is also caused by vaccination because it is a proven result of inflammation and vaccination incites the inflammatory response on purpose in order to induce antibodies.  In fact, within days of my BCG vaccination I was suicidal and self-harming despite never having had a history of depression before and being a previously happy teenager, then I disended into frequent fevers, dizziness, panic attacks, ear infections, flu like symptoms, burst ear drums and then finally, hyperacusis.

In fact, scientists have confirmed that inflammation causes depression in animals and guess what they used to induce it in 2009?  BCG vaccine.  But don’t worry, they assured people, it’s not one we use in the US.  Well, it was one they used in the UK and other countries.

‘Scientists have confirmed the role of an immune-activated enzyme in causing inflammation-related depression-like symptoms in mice. The work clarifies how the immune system can trigger depression and, more broadly, demonstrates the potential of this animal model for exploring the relationship between chronic inflammation—a common feature of diseases such as heart disease, cancer, and diabetes—and depression.

When an individual is infected with viruses or bacteria, cells of the immune system respond by secreting proteins called cytokines. These cytokines not only trigger inflammation and orchestrate the body’s immune response against the infection, but they also cause changes in behavior, such as fatigue and withdrawal. Beyond these commonly experienced behavioral signs of illness, previous research has shown that cytokines can also cause depression in people with physical illnesses but who have no prior history of mental illness. For instance, around one-third of patients receiving the cytokine interferon-α for treatment of cancer or hepatitis C develop major depression. Clinical evidence has suggested that an enzyme (IDO) activated by these same cytokines might be a key player.

In this work, scientists used a weakened form of the tuberculosis relative, bacille Calmette-Guérin (BCG), to model chronic inflammation. This strain of bacteria is used outside the U.S. as a vaccine for tuberculosis. Infection of mice with high doses of BCG persistently activates the immune system; as a consequence, the mice develop depressive-like behavior after initial signs of illness have subsided. ‘


So that could also explain why I also struggled with depression for years.  I know I was chronically inflammed.  I could feel nerve pain down both sides of my neck and behind the ears, which would get worse the with the more noise I was exposed to.  It felt as if my nerves were inflammed and my ears would go bright red in colour, but for years I was never believed, with doctors telling me it was muscle pain (I do know the difference between muscle and nerve pain).  In the years after my initial recovery from hyperacusis, I would get relapses, always accompanied by nerve pain at the sides of my neck.  When the neck nerve pain stopped, my hearing tolerance would return to normal.  Eventually I got a GP to believe me and he prescribed ibuprofen (an anti-inflammatory) that I had to take for ten weeks before I recovered from hyperacusis.  Each time I re-lapse, it is anti-inflammatory medication that reduces my symptoms.

Web MD say that sensory nerve damage may cause pain and sensitivity (both symptoms of hyperacusis – a sensory processing disorder).  They also say:

Drug side effects and toxic substances. Various substances that are taken into the body intentionally or unintentionally have the ability to cause nerve pain and nerve damage. These include medications, such as chemotherapies for cancer and certain drugs used to treat HIV. Toxic substances that may be ingested accidentally, including lead, arsenic, and mercury, may also cause damage to your nerves. ‘ –

Notice their reference to mercury, of which thimerosal is 49% mercury, and that’s not including the many other toxic substances in vaccines.

Furthermore, another study done in 2009 found that repeated vaccination caused auto-immune disease in otherwise normal animals and it also caused tissue damage, something I know and can feel that I have:

‘Repeated immunization with antigen causes systemic autoimmunity in mice otherwise not prone to spontaneous autoimmune diseases. Overstimulation of CD4+ T cells led to the development of autoantibody-inducing CD4+ T (aiCD4+ T) cell which had undergone T cell receptor (TCR) revision and was capable of inducing autoantibodies. The aiCD4+ T cell was induced by de novo TCR revision but not by cross-reaction, and subsequently overstimulated CD8+ T cells, driving them to become antigen-specific cytotoxic T lymphocytes (CTL). These CTLs could be further matured by antigen cross-presentation, after which they caused autoimmune tissue injury akin to systemic lupus erythematosus (SLE).

Immunization of mice 12× with OVA led to re-expression of the V(D)J recombinase complex and enlargement of the spleen (Figure S4A), and an increase in anti-dsDNA antibody, which is uniquely linked to autoimmune tissue injury in lupus nephritis [14] (Figure S2A). Pathological findings included diffuse membranous (wire-loop) and/or proliferative glomerulonephritis in the kidney (Figure 3A), infiltration of plasma cells around hepatic bile ducts (Figure S4B), enlarged lymphoid follicles with marked germinal center in spleen (Figure S4B), occasional lymphocyte infiltration into the salivary glands (data not shown), lymphoid cell infiltration into the thyroid, and perivascular infiltration of neutrophils and macrophages into the skin dermis of the auricle (Figure S4B). The lupus band test, diagnostic of SLE, was positive in the skin at the epidermal-dermal junction (Figure 3B).’ –

I was 95% recovered after 11 years of constant, total suffering (and not able to work) by pink noise brain re-training with the ENT unit, no more vaccines ever again, vitamin therapy, whole food diet and detox.  When I re-lapse I have to have anti-inflammatories to recover myself again.  I have never been 100% recovered and I have multiple inflammatory illnesses that I now HAVE to take medications for.  In all likelihood, I will probably be on anti-seizure meds (that numb malfunctioning nerves) and anasethetics for the rest of my life, and all because of a vaccine that didn’t work, for a disease I wasn’t going to get (TB is caused by over-crowding, unsanitary conditions which did not apply to me).

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